It has been “hypothesized” a Western Diet characterized in part by excessive sugar consumption. Maybe a factor for increased cancer incidences. Whereas, in contrast t0 African countries that principally consume foods high in thiamine have reduced cancer rates. It’s hard to say anyone nutrient lacking is the root cause when instead it takes a natural balance (cancer is a survival mechanism not a disease?)
Although no direct study has been evaluated, the dietary intake of thiamine and cancer risk have provided conflicting results. Thiamine (or Vitamin B1) deficiencies have been found in many sugar abusers, which causes abnormally slow oxidation in bodily cells. With also increased consumption of sugar…. colds, tonsillitis, sinus infections, catarrhal disease and cancer is on the raise.
People over time can become allergic to sugar, producing internal yeast over growth and its toxic by-product from consuming excessive sugar causing allergies and in some cases cancer. Excessive sugar can change the body’s biochemistry…. so the adrenal glands are unable to cope satisfactory with the toxic state of being.
While in a sugar glutton when (s)he is exposed to germs, his/her resistance is lowered and the Bacillus cell ( fungus) grows more rapidly in his/her body. It has long been known a good nutritious diet goes far in helping the individual recover from disease. In the fore mention, first paragraph, before sugar can cause a thiamine deficiency which in turn leave your body exposed to all types of health maladies.
A 2008 study, examined the relationship between the intake of B – vitamins and incidences of breast, endometrial, ovarian, colorectal and lung cancer in women. Interestingly, reduced thiamine levels also increased the number of aberrant crypt foci ( fungus) in the colons of rats fed a sucrose – based diet. Patients with severe malnutrition have exhibited Baker’s cyst, osteosarcoma and submandibular cyst.
Which was cured without recurrence after thiamine administration, which suggest a role of thiamine deficiency in tumor development. A limited number of studies and case reports have determined the over all status in cancer patients. Clinically, thiamine status is quantified biochemically using a TKT assay of white blood samples.
This assay…. which involves measuring the increase in the activity of the thiamine – dependent enzyme TKT after added TPP. If Deficient in thiamine, exogenous TPP will stimulate TKT activity, termed also the TPP effect. Similarly, this increased TPP effect was characterized in patients (B – Chronic Lymphocytic leukemia, Burkett’s Lymphoma, and Accute Myelomocytic Leukemia.)
Although the reason for a decrease in thiamine states in the blood is unclear. One study, also noted cancer patients had a higher level of thiamine urinary excretion. The authors of this study suggest that the thiamine status might not be due to reduced dietary intake of thiamine, but an inability to activate thiamine to TPP.
This reduction in peripheral thiamine/TPP may be a consequence of extensive accumulation and/or utilization by cancer cells, During tumor growth, cancer cells maintain a constant level of TPP, while the host liver tissue exhibited a perpetual decline. Overall, these studies strongly suggest a preferential accumulation of thiamine into cancer cells. That may be responsible for the alteration in peripheral thiamine status during malignancy.